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MT-1 is a linear tridecapeptide closely matching the full -MSH sequence with Nle4/D-Phe7 modifications preferentially activating MC1R with limited central melanocortin receptor activity at research doses

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Different mechanisms including DNA damage, promotion of lipid peroxidation, and protein modification are caused by oxidative stress lead to tissue damages which have been implicated in the pathogenesis of various systemic diseases

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