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doi: 10.1128/JVI.02199-08

Protein arginine methyltransferase 1 coactivates NF-kappaB-dependent gene expression synergistically with CARM1 and PARP1

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In patients with FAOD, reduced or loss of function of one of the mitochondrial proteins involved in fatty acid transport or metabolism can lead to tissue accumulation of fatty acids and/or their intermediate metabolites, as well as metabolic decompensation due primarily to the resulting depletion of tricarboxylic acid cycle intermediates such as acetyl-CoA [12]

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