doi: 10.1016/j.heliyon.2020.e05469 239 HuangYCaoSZhangQZhangHFanYQiuFet al
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For BPC 157-treated rats, in early and in late periods, the level of oxidative stress in the liver (MDA- and NO-levels, otherwise known to be increased and critical mediators of liver fibrosis[25]) was almost continuously within normal values as it was in rats with bile duct occlusion[25]
This move is in an attempt to prevent cachexia, which accounts for 20% of cancer mortalities (5)