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In line with our short-term metabolic analyses, previous studies in New Zealand obese and diet-induced obese male mice suggested that GLP1/E acts primarily at central levels to reverse metabolic alterations by decreasing food intake and body weight, while the potential beneficial effects on pancreatic beta-cells were possibly secondary and indirectly-mediated 33,48

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Conversely, another study that demonstrated glutamine supplementation was unable to improve the growth performance of Atlantic salmon parr under high temperature regimes, while recording an increase in pyloric caeca surface area [63]

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