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Blumenreich M, Andreeff M , Murphy ML, Young C, Clarkson B

This hypothesis was subsequently validated in experiments using RAW264.7 mouse macrophage-like cells and in mice infected with Bacillus Calmette-Gurin (BCG)

NK cells accumulate in the diseased choroid and inhibit pathological changes in nvAMD by promoting NETosis and NET production by neutrophils (201, 202)

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Given that we have established selenoproteins such as GPx3 and SELENOP to be substantially reduced in those affected with disease using multiple applications and cohorts, perhaps a reduction in the ability to effectively manage oxidative damage is a leading factor for disease susceptibility and severity, when all other risk factors have been met, which are typically present even in those without konzo

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