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These interactions determine PRMT5's enzymatic activity toward Sm proteins and histones [102]

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For example, PEPCK and G6P activities are increased in obese individuals (Ropelle et al., 2009

Interestingly, depletion of mitochondrial GSH has also been associated with apoptosis or ATG induced by chemotherapeutic drugs (Samudio et al., 2005

Moreover, both insulin and PE also activates P38 in skeletal muscle, an vital molecule to activate PGC1- downstream pathway (see Figure 7) to promote mitochondrial remodeling resulting in improvement in glucose and fat metabolism (Hood et al., 2015)

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