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doi:10.2174/1874473711003020103 Pittler MH, Verster JC, Ernst E

In pediatric rhabdoid tumors (RTs), which often lack targetable mutations, a combined high-throughput drug screen and genome-scale CRISPR-Cas9 knockout study identified receptor tyrosine kinases (RTKs) and the downstream signaling protein SHP2 (encoded by PTPN11) as essential for tumor survival despite the absence of genetic alterations, highlighting non-mutational dependencies [118]