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Decades of peer-reviewed literature have characterised its role in redox homeostasis, glutathione-S-transferase-mediated xenobiotic conjugation, reactive-oxygen and reactive-nitrogen species buffering, hepatic detoxification biology, and protein S-glutathionylation as a redox-signalling post-translational modification

The toxicity of free radicals contributes to the damage of cellular macromolecules (such as DNA, proteins and lipids), inflammation, tissue damage and subsequent cellular apoptosis (Reference Ames, Shigenaga and Hagen14)

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