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D4F prophylaxis enables redox and energy homeostasis while preventing inflammation during hypoxia exposure

Neubauer ZMMO, Singal A, Lipner SR

Gene ontology analysis revealed that many components of the mitochondrial translation machinery are downregulated in response to GSH depletion, a phenotype previously linked to electron transport chain dysfunction 14 (Fig

All genes were expressed in neurons of the nodose ganglion (Fig