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Journal of Applied Physiology (2011) 4

Results We show that our model accurately reproduces published clinical and experimental data on the dose-dependent time course of acetaminophen in the plasma, the accumulation of acetaminophen and its metabolites in the urine, and the depletion of glutathione caused by conjugation with the toxic product

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Various microbial metabolites have been reported to influence these neuroprotective pathways in multiple beneficial ways

doi:10.1007/s10522-025-10315-x PMC:PMC12411320 (note: a correction was published 15 November 2025, doi:10.1007/s10522-025-10326-8, replacing Figs

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