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After two weeks with no adverse effects, increase to the standard dose

Both compounds require subcutaneous administration for bioavailabilityoral forms degrade rapidly in gastric acid and hepatic first-pass metabolism

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GHK-Cu blocks ferritin channels and the release of tissue-damaging free (oxidative) iron after injury, thus blocking iron-catalyzed lipid peroxidation that often occurs after injury and interleukin-1 damage to pancreatic islet cells