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At week 30 the primary HbA1c endpoint was missing for 7%, 5% and 5% of patients and during the trial rescue medication was initiated by 14%, 2% and 1% of patients randomized to placebo, OZEMPIC 0.5 mg and OZEMPIC 1 mg, respectively

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doi: 10.1007/s00125-023-06053-9

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The ability of lipids to induce CCK release and activate VANs to lower food intake may be due to chylomicron formation and subsequent activation of sensory machinery on the basolateral side of EECs, as mentioned previously