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Frequency of injections will vary

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Normal -cells can compensate for insulin resistance by increasing insulin secretion or -cell mass, but insufficient compensation leads to the onset of T2DM

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Activation of GLP-1R suppresses food intake and induces postsynaptic depolarization of membrane potential (Figure 1A) and/or presynaptic modulation of glutamatergic or GABAergic neurotransmission (Figure 1B)