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GABA is, similarly to glutamate, after cerebral ischemia, released from neurons and also microglia, and either reabsorbed by neurons or uptaken by surrounding glial cells for further metabolism (Fig

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IL-12 is primarily synthesized by immune cells such as dendritic cells and macrophages, and it plays a crucial role in driving Th1 cell differentiation, stimulating activation of T, B, and NK cells, and inducing the reprogramming of immunosuppressive cells [134]

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