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Within this model, curcumin does not substitute for established antidiabetic pharmacotherapy whether metformin, sodium-glucose cotransporter-2 (SGLT2) inhibitors, glucagon-like peptide-1 (GLP-1) receptor agonists, or insulin but rather complements standard treatment by targeting residual inflammatory burden, oxidative stress, and metabolic dysregulation that persist despite conventional management

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Analyses of the recycling receptor, FcRn, in live cells reveal novel pathways for lysosomal delivery