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Uric acid attenuates nitric oxide production by decreasing the interaction between endothelial nitric oxide synthase and calmodulin in human umbilical vein endothelial cells: a mechanism for uric acid-induced cardiovascular disease development

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Because in most of these studies, patients in the higher versus lower Kt/V (or URR) groups were dialyzed with membranes of different composition and flux, the role of dialysis dose cannot be separated from the effects of membrane permeability or biocompatibility

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