A rapid reduction in serum uric acid during urate-lowering therapy can destabilize crystal surfaces and trigger the release of proinflammatory substances
Diffusion tensor imaging shows reduced fractional anisotropy in white matter tracts ( In pediatric populations, congenital hypothyroidism and untreated iodine deficiency remain major causes of intellectual disability worldwide ( 3.3 Hyperthyroidism and neurological hyperactivity syndromes Hyperthyroidism, in contrast, produces a state of neural hyperexcitability ( Concurrently, T3 upregulates L-type calcium channels while reducing buffering proteins such as calbindin, creating a vulnerability to excitotoxicity ( 3.4 Thyroid-specific oxidative stress and neuroinflammation Oxidative stress and neuroinflammation are key mechanisms linking thyroid dysfunction to neuronal injury
We investigated whether this regulation was dependent on modulation of DGAT2 and GPAT4 targeting the LD surface
Sjogren K, Jansson JO, Isaksson OG, Ohlsson C
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