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The researchers constructed a model that introduced the ALAS2 missense mutation erythroblasts derived from human cord blood, in which enhanced BACH1 expression was emerged, leading to increased susceptibility to ferroptosis (Ono et al., 2022)

It is not part of a larger complex but interacts with other antioxidants to protect cells from damage

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Targeting glutamine metabolism inhibitors to tumor tissues but not healthy cells may circumvent these drawbacks