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33 Here, we aimed to uncover therapeutically exploitable PDAC subtype-dependent metabolic vulnerabilities that evolve with indirect dual interference with RAS pathway activity via vertically combined SHP2/MEK1/2 inhibition
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A more recent study demonstrated that the delivery of instability-prone Mecp2 ( iMecp2 ) transgene cassette using an adeno-associated virus (AAV) vector in symptomatic Mecp2 mutant mice significantly improved locomotor activity, lifespan and normalized gene expression [44]
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