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Fig 3E summarizes the GLP-1 signaling dependent and independent control of LAG-1 peak accumulation, indicating that about one third of LAG-1 accumulation is promoted by GLP-1 transcriptional targets lst-1 and sygl-1

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Thus, the expression of downstream NOX4/ROS/VEGF signaling pathway-related proteins is activated and the proliferation of breast cancer cells is increased, whereas the proliferation of breast cancer cells is inhibited in the control group with the addition of inhibitors

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