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MOTS-c is a mitochondrial-derived peptide that activates AMPK through direct mitochondrial signaling and nuclear translocation under metabolic stress, whereas metformin activates AMPK indirectly by inhibiting Complex I of the mitochondrial electron transport chain, and AICAR mimics AMP to trick the cell into AMPK activation

Bruno JG (2022) Successes and failures of static aptamer-target 3D docking models

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PMID: 27671008

The quality of your solvent directly affects the quality of your reconstituted peptide