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Combining a systemic bone-building signal with a compound that may enhance local blood supply and cellular recruitment at the fracture site is a compelling research avenue

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Protein interacting with C kinase 1 (PICK1), which binds to the GluR2 via the PDZ domain [37], provides binding domains for PKC and glutamate receptor-interacting proteins (GRIP), which ultimately helps in phosphorylation and the internalization of AMPAR [37,38,39]

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Moreover, integrating nanotechnology into personalized medicine allows treatment strategies to be tailored to individual patient needs (Germain et al