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These findings underscore the urgent need for targeted antioxidant therapies capable of quenching this systemic ROS burden and restoring metabolic homeostasis

Mechanism of arylating quinone toxicity involving Michael adduct formation and induction of endoplasmic reticulum stress

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In Vivo Protection: Pretreatment with LASSS significantly reduced HGF nitration in a tail-suspension mouse model of disuse muscle atrophy

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Patients with congenital abnormalities were excluded