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Along these lines, increased duodenal contractility by the ENS is characteristic of T2D and likely contributes to metabolic dysfunction

Coexpressed nitric oxide synthase and apical beta(1) integrins influence tubule cell adhesion after cytokine-induced injury

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591 Additionally, boosting NAD + levels via inhibiting ACMSD (an enzyme restricts the de novo synthesis of NAD + from tryptophan) also protects against AKI after renal I/R injury