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What could that be? In a way, your study found GLP-1s are, at times, working too well

For instance, exosomal miR-140-5p suppresses SLC7A11 expression, impairing the system xc-/GSH/GPX4 axis, reducing GSH synthesis, and promoting cardiomyocyte ferroptosis and cardiac injury [101]

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When cells were treated with lysosomal reagents (e.g., ammonium chloride) or lysosomal protease inhibitors (e.g., chloroquine), the degradation of LC3-II was blocked, resulting in the accumulation of LC3-II

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These issues might lead to permanent vision loss or long-term eye pain