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Simultaneously, the IRAK-TRAF6 complex activates mitogen-activated protein kinase kinase (MKK) cascades, thus leading to activation of activator protein 1 (AP-1), which is another critical transcription factor in inflammation (50)

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The mechanism behind these effects involves GHK-Cu's ability to stimulate both the synthesis and breakdown of dermal proteins, essentially accelerating the entire wound healing process [1]

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doi: 10.3389/fmicb.2023.1137083 158 SunY.RevachO