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These polymorphic variants usually have decreased enzyme activity and are often reported with reduced enzyme stability as well as disruptions in protein folding ( EpidemiologyGlobal and Local Insights The global prevalence of G6PD deficiency was estimated to be 4.9% ( Since its discovery in 1956 by Carson et al
The development of an optimal therapy is, however, complicated by certain side effects, including the possibility of post-treatment hypothyroidism and recurrence of hyperthyroidism following an initial successful treatment
This debate is reflected in a number of animal models and several human diseases, including careful analysis of human MS (Figure 3
