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The findings provided a proof of concept for this therapeutic strategy by demonstrating the efficiency of clorgyline, rasagiline, and selegiline in inhibiting MAO and decreasing endogenous DOPAL synthesis [40]

Mechanistic studies examine its interactions with nitric oxide signaling, prostaglandin pathways, and gastrointestinal dopamine and serotonin turnover, including their relevance to experimental models of intestinal inflammation and gastrointestinal dysfunction

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B.KoskinenK.PaulinL.PekkonenE.et al

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