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We performed joint factorization of the transcriptomic and global proteomic data using multiomic factor analysis (MOFA) 27 and found that MOFA factors 1 and 2 captured the primitive versus committed and NPM1 mutation status (Extended Data Fig

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However, other pathophysiological modifications within melanoma cells may contribute to therapy resistance [4, 5], demanding for large-scale computational models to simulate the complex physiology of a tumour in an integrated manner and comprehensively foster the identification of alternative therapeutic vulnerabilities applying high throughput in silico approaches [6, 7]