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Non-Medicinal Ingredients: silicone dioxide

Using VSMCs stimulated with oxidized LDL as a model of atherosclerosis, it was shown that TUG1 silencing increased miR-148b, leading to a decline in IGF-2 which was found to be a target of miR-148b [78]

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Uetrecht and Naisbitt 2013)

The authors of this study explain the upregulation with the increased availability of Sec, although this was not shown for knockout of other selenoproteins, e.g., GPX1 [107]

Different forms exist (capsule, sublingual, solution), but no official dosing guidelines are established due to limited studies