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[4] Synaptic plasticity in both excitatory and inhibitory synapses has been found to be dependent upon postsynaptic calcium release

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In a study involving rats with hind limb ischemia, BPC-157 treatment was found to increase the expression of VEGFR2, subsequently activating the VEGFR2-Akt-eNOS pathway

Higher doses may increase gastrointestinal side effects without improving outcomes