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Therefore, to further dissociate the effects of GSH and NRF2 on lipid abundance and assess if NRF2 activation alone mimics the effects of liver-specific Gclc deletion, we treated Gclc WT mice with CDDO-methyl (CDDO-Me), a form of bardoxolone with increased bioavailability

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Epithelial cells of the tubular and collecting ducts were enriched in the second band across the 7%/11% iodixanol interface (designated 1.045-1.063 g/ml), while non-tubular cells were enriched in the fourth band across the 11%/16% iodixanol interface (designated 1.073-1.091 g/ml)

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