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Protective effect of N -acetyl-l-cysteine on amyloid [beta]-peptide-induced learning and memory deficits in mice

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Since oxidative inactivation of peroxiredoxin was shown to be correlated with activation of c-Jun N -terminal protein kinase (JNK) and/or p38 kinase [70, 71], we expect the increased activation of JNK and p38 kinase in MDMA-exposed tissues

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