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In the case of eWAT, an increase in endogenous fatty acids following adherence to a HFD activates two sensors within adipocytes, Toll-like receptor 4(TLR4) and inflammasomes, which causes an influx of neutrophils to enter the eWAT, leading to increased levels of chemokine and cytokine expression (132)

( a , e , i ) Representative blots are shown

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Introduction of shRNA to knock down ACTL6A expression inhibited the wound healing and metastatic ability of GC cells (Fig

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