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487,488 Increased ECM rigidity worsens the functions of senescent cells by boosting the secretion of matrix metalloproteases, intensifying ECM damage, and triggering pathways that promote senescence, fibrosis, and inflammation

The mechanism of action of DSePA against RP involved its ability to suppress the radiation-induced sustained inflammatory response by upregulating the pulmonary activity level of GPx and concurrently blocking the NF-kB/IL-17/G-CSF/neutrophil axis (Table 2) (Fig

[1] [2] Its reduced (NADH) and phosphorylated forms (NADP+ and NADPH) are as important as NAD+

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F., Oztop, D

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