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However, recent work has shown that, in addition to using histones as substrates for deacetylation, HDACs can also inhibit the acetylation of non-histone proteins and modulate diverse neurobiological processes [59, 60]

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"Viral mediated expression of insulin-like growth factor I blocks the aging-related loss of skeletal muscle function." Proceedings of the National Academy of Sciences, 95(26), 15603-15607