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We found that blocking the translation of NRF2 prevented GSH accumulation ( Figure 7F ) as well as negated the immunosuppressive action of melatonin treatment ( Figures 7H, I )

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The dissection of the multiple mechanisms at the basis of c-MET pathway activation and primary and acquired resistance to c-MET inhibition in different tumor types will lead to further improvement in targeted drug development and patient selection to optimize outcome and toxicity exposure

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