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According to the findings related to the three aforementioned metabolic pathways, in addition to those showing iron accumulation, lipid peroxidation, and redox stress, it is not difficult to infer that ferroptosis plays a vital role in the pathogenesis and progression of IgAN

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IGF-1R inhibition enhances radiosensitivity and delays double-strand break repair by both non-homologous end-joining and homologous recombination