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doi:10.4239/wjd.v6.i14.1274 Minich DM, Brown BI

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in vitro experiments demonstrated that SGK1 knockdown attenuated erastin-induced downregulation of Slc7a11, GPX4, GSH, and oxidized glutathione (GSSG), while reducing lipid peroxidation, iron accumulation, and mitochondrial damage upregulation, suggesting SGK1's critical role in CHD-associated ferroptosis (44)

Consistently, we observed that knockdown of TFRC robustly protected against ferroptosis (Supplementary Fig