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Abstract Because reactive oxygen species (ROS) have been implicated as mediators of inflammatory bowel disease (IBD), the purpose of the present work was to determine the functional role of mucosal GSH in the trinitrobenzenesulfonic acid in 50% ethanol (TNBS+ethanol)-induced colitis in rats

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This pro-angiogenic effect is one reason BPC-157 can reverse liver damage rather than merely slow its progression

Under oxidative conditions, cysteine residues on proteins can form mixed disulfides with GSH

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