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APOM, by binding sphingosine-1-phosphate (S1P), not only triggers anti-inflammatory signaling via S1P receptors but also suppresses inflammation by inhibiting NF-B nuclear translocation through the PI3K/AKT pathway [55, 56]

This peptide has been shown to have a profound effect on biomarkers of aging, including the elongation of telomeres, which are essential for cellular reproduction and longevity

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Amy Arnstens group showed that it acts on the dendritic spines of prefrontal neurons by closing HCN ion channels, raising the gain of the networks that hold information in mind while the drug is present

DSIP is investigated as a peptide signal that may fine-tune endogenous regulatory circuits

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