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they are responsible for approximately 90% of the incretin effect [34]

These findings suggest that GLP-1 receptor agonists, particularly at higher doses, may offer disease-modifying benefits beyond symptomatic improvement

L.AndradesA.Von BergenM.et al (2016)

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Additional modulators, including Acyl-CoA Synthetase Long-Chain Family Member 4 (ACSL4), which promotes PUFA incorporation into membrane phospholipids, and lipoxygenases (LOXs), further influence ferroptosis sensitivity by regulating lipid composition and oxidation dynamics (90, 91)