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Diabetologia 68, 11151125

Neither glutathione trisulfide (GSSSG, a potent anti-oxidant) nor lipoic acid enhanced c-met binding or nitration resistance, and thus served as controls

In support to this notion, we have shown: (i) L-cysteine, an activator of H 2 S producing enzymes (CSE and CBS), or the exogenous H 2 S donors NaHS and GYY4137 inhibited OA-induced GLP-1 and PYY release, and (ii) inhibition of release is due to suppression of the Epac/PLC-/Ca 2+ pathway downstream of cAMP pathway

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This genetic insight should be interpreted alongside biomarker assessment and clinical evaluation by your qualified healthcare provider