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MOTS-c prevents weight gain and reduces adiposity in ovariectomized mice (used to mimic menopause) by reducing blood lipids and hepatic triacylglycerol levels, while also enhancing lipolysis [44]

Considering that Keap1 can also direct Nrf2 nuclear export [29], it remains to be elucidated whether p65/Keap1 interaction and subsequent Nrf2 inactivation occurs in the nucleus or in the cytoplasm

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The Cambridge paper specifically flagged this as a critical gap in the research that future trials need to address, because no studies have yet systematically tracked body composition changes after discontinuation