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Both are naturally produced by your body, but as we age, our internal levels of these vital compounds begin to decline

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We found that internalized exendin-phe1-FITC was situated in the least, and exendin-asp3-FITC in the most, acidic endosomes (Supplementary Fig

In contrast, high glucose-induced GLP-1 release was unaffected by PPAR/ inhibition, suggesting that C3G-induced PG gene transcription to increase GLP-1 levels in STC-1 cells was specifically mediated through the PPAR/--catenin-TCF signaling pathway

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