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PMID: Summary Keywords GLP-1 receptor (GLP-1R), metabolic dysfunction, Mitochondria-ER Contact Sites (MERCS), signalosomes, GLP1 receptor agonist (GLP1-RAs) Citation Cruz P, Puebla-Huerta A, Troncoso MF, Silva-Pavez E, Hidalgo-Fadic Y and Ahumada-Castro U (2026) GLP-1 receptor and Mitochondria-ER Contact Sites: an emerging mechanism of metabolic regulation

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[1] [2] B12 functions as a cofactor in cellular metabolism, participating in the conversion of homocysteine to methionine and the metabolism of methylmalonic acid, both crucial for energy production

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Any new or worsening neurological symptoms warrant thorough clinical evaluation to distinguish between medication effects, disease progression, or unrelated conditions