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CDK1 directly binds and phosphorylates the S447 site of ACSL4, promoting the ubiquitination degradation of ACSL4, while genetic and pharmacological blocking of CDK1 enhances ACSL4 activity, promoting ferroptosis of colon cancer cells and increasing their susceptibility to oxaliplatin

Statements Data availability statement The raw data supporting the conclusions of this article will be made available by the authors, without undue reservation

The NOX family of proteins is primarily localized to the plasma membrane