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The Gln metabolism in PCa is tightly controlled by well-described oncogenes such as MYC, AR, and mTOR

Zhang, Y., Li, X., Zou, D., & Ren, G

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They may include increased GSH utilization upon exposure to oxygen, insufficient GSH synthesis due to immature synthesis enzymes or to reduced availability of cysteine, the rate-limiting precursor amino acid for GSH synthesis, or impaired regeneration of oxidized glutathione (GSSG) to GSH

Shortening this interval increases peak drug concentration and the risk of gastrointestinal side effects

This dietary intervention can impact metabolic flux in one-carbon metabolism, inhibit tumor growth, and sensitize tumors to chemotherapy and radiotherapy in certain human cancer cells in a tumor-cell autonomous manner 14