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In the pathological microenvironment of COPD, lipid metabolism undergoes significant reprogramming, characterized by FAO abnormalities, dysregulated sphingolipid and glycerophospholipid metabolism, and imbalanced regulation of triacylglycerol (TAG) and phospholipid synthesis

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Studies have described high growth of different species of Clostridium and Candida as well as members of the genus Sutterella [89-91]

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