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The IGFBPs have a central role in modulating the oncogenic actions of IGFs mediated by IGF1R signaling, but they also have well-documented functions that occur independently of IGF- and IGF1R-dependent pathways [17]

This also helps explain why cortisol and GLP-1 are often discussed together in metabolic health research, since stress biology may influence appetite signaling, inflammatory tone, and endocrine responsiveness

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These GLP-1 agonist medications may support appetite regulation, promote satiety, and be included in broader provider-directed metabolic health strategies

Int J Oncol (2015) 47(1):35360